Semax 10mg
Nootropic peptide for cognitive enhancement and neuroprotection
In Stock (25 available)
SKU: ML-SEMAX-10MG
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Buy 3+ vials = 10% off · Buy 5+ vials = 15% off
Description
Semax 10mg
Semax is a synthetic peptide derived from ACTH (adrenocorticotropic hormone). Developed in Russia, it is approved there as a nootropic and neuroprotective agent.
Key Benefits
- Enhances BDNF (Brain-Derived Neurotrophic Factor) expression
- Improves memory, focus, and cognitive performance
- Neuroprotective properties
- Supports learning and mental clarity
- Well-studied safety profile
Research Highlights
- Approved in Russia for cognitive disorders
- Studied for stroke recovery and cognitive enhancement
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Research References
The Potential of the Peptide Drug Semax and Its Derivative for Correcting Pathological Impairments in the Animal Model of Alzheimer's Disease.
Evaluates Semax as a therapeutic agent for Alzheimer's disease in animal models.
The Effect of Peptide Semax, an ACTH(4-10) Analogue, on Intracellular Calcium Dynamics in Rat Brain Neurons.
Investigates Semax's mechanism of action on neuronal calcium signaling.
Semax peptide targets the mu opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury.
Reveals Semax promotes spinal cord injury recovery through mu opioid receptor gene targeting.
Genes That Associated with Action of ACTH-like Peptides with Neuroprotective Potential in Rat Brain Regions with Different Degrees of Ischemic Damage.
Identifies genes associated with neuroprotective action of ACTH-like peptides including Semax in ischemic brain regions.
ACTH-like Peptides Compensate Rat Brain Gene Expression Profile Disrupted by Ischemia a Day After Experimental Stroke.
Shows Semax restores disrupted gene expression in rat brains following experimental stroke.
Changes of Transcriptomic Activity in Rat Brain Cells under the Influence of Synthetic Adrenocorticotropic Hormone-Like Peptides.
Characterizes transcriptomic changes induced by Semax in rat brain cells.
Synthetic Adrenocorticotropic Peptides Modulate the Expression Pattern of Immune Genes in Rat Brain following the Early Post-Stroke Period.
Shows Semax modulates immune gene expression in the brain during the critical early period after stroke.
Effect of ACTH4-10Pro8-Gly9-Pro10 on anti-inflammatory cytokine expression in acute spinal cord injury models.
Studies Semax's effect on anti-inflammatory cytokine expression in spinal cord injury.